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Figure 1: (a) External surface of the heart in a 31-year-old male with alcoholic cirrhosis showing significant increase in epicardial fat (RV right ventricle, LV left ventricle); (b) Opened out LV inflow tract showing a uniform layer of adipose tissue all around (LA left atrium, MV mitral valve); (c) Close-up of the anterior free wall showing adipose tissue insinuation (arrows) into the myocardium; (d) Small groups of myofibers separated by adipocytes (Hematoxylin and eosin ×250); (e) Deposits of fat below a thickened fibrotic endocardium (Hematoxylin and eosin ×250)

Figure 1: (a) External surface of the heart in a 31-year-old male with alcoholic cirrhosis showing significant increase in epicardial fat (RV right ventricle, LV left ventricle); (b) Opened out LV inflow tract showing a uniform layer of adipose tissue all around (LA left atrium, MV mitral valve); (c) Close-up of the anterior free wall showing adipose tissue insinuation (arrows) into the myocardium; (d) Small groups of myofibers separated by adipocytes (Hematoxylin and eosin ×250); (e) Deposits of fat below a thickened fibrotic endocardium (Hematoxylin and eosin ×250)